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mRNA in Breast Cancer

Explore this research category across breast cancer datasets

142
TOTAL MRNAENTRIES
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142 research entries found

MRNA
RNA Name Mechanism of Action Effect on Cancer Cancer Type Reference
CSNK2A1

mRNA
Casein kinase II alpha (CK2?) phosphorylates targets to promote survival signaling and suppress apoptosis. Overexpressed in breast cancer (including TNBC); CK2 inhibition induces apoptosis. Breast Cancer PubMed
Reference

PMID: 36114992
CASP9

mRNA
Initiator caspase of the intrinsic (mitochondrial) apoptosis pathway activated by Apaf-1 in the apoptosome; activates executioner caspases like caspase-3. CASP9 expression associates with prognosis in breast cancer; restoration of caspase-9 can suppress metastatic behavior in TNBC models in recent studies. Breast Cancer PubMed
Reference

PMID: 36199443
PDCD10

mRNA
Scaffolding component (CCM3); can promote apoptosis through stress kinases in some contexts. Loss increases chemoresistance in breast cancer, suggesting a role in apoptosis and therapy response. Breast Cancer PubMed
Reference

PMID: 30408026
BCL2L14

mRNA
BCL2L14 (BCL-G) is a BCL-2 family member with pro-apoptotic BH3-like activity in certain contexts, promoting mitochondrial apoptosis when active. BCL2L14 fusions/alterations have been observed in aggressive breast cancers; linked to therapy resistance features. Breast Cancer PubMed
Reference

PMID: 32321829
CASP2

mRNA
Caspase-2 acts as a stress-response initiator caspase involved in DNA damage-induced apoptosis and can function upstream of mitochondrial pathways. Altered CASP2 expression associates with prognosis in breast cancer/TNBC; functional context varies by study. Breast Cancer PubMed
Reference

PMID: 37093683
CCAR2

mRNA
Inhibits SIRT1 to stabilize p53 and promote p53-mediated apoptosis; also coactivates ER?/PEA3 under some contexts. Dual role in breast cancer; linked to prognosis and can modulate apoptosis in BC. Breast Cancer PubMed
Reference

PMID: 37524873
BCL2L1

mRNA
BCL2L1 (BCL-xL) is an anti-apoptotic Bcl-2 family protein that sequesters pro-apoptotic BH3-only proteins and Bax/Bak to prevent MOMP and apoptosis. Overexpression in TNBC contributes to chemoresistance; BCL-xL inhibition enhances apoptosis and chemosensitivity. Breast Cancer PubMed
Reference

PMID: 38898061
CASP10

mRNA
Initiator caspase in extrinsic apoptosis (death receptor pathway). Expression observed in TNBC; functional studies limited in last 10 years but expression associated with apoptosis responses. Breast Cancer PubMed
Reference

PMID: 40806590
HIF1A

mRNA
Hypoxia-activated HIF-1? transcriptionally induces BNIP3/NIX to promote mitophagy and hypoxia-induced autophagy. High HIF-1?/BNIP3-axis activity associates with aggressive TNBC biology; BNIP3 loss (with hypoxia/HIF-1? signaling) promotes TNBC progression and metastasis. Breast Cancer PubMed
Reference

PMID: 26232272
BNIP3

mRNA
HIF-1?�induced mitophagy receptor; promotes BNIP3/NIX-dependent mitophagy and hypoxia-induced autophagy. BNIP3 loss promotes TNBC progression and metastasis under hypoxia; BNIP3 axis is clinically relevant in TNBC. Breast Cancer PubMed
Reference

PMID: 26232272
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